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Evaluation of [18F]FD4 PET targeting alpha synuclein in Parkinson Disease and Multiple System Atrophy

Evaluation of [18F]FD4 PET targeting alpha synuclein in Parkinson Disease and Multiple System Atrophy

FD4 is a new investigational radiotracer or imaging agent that binds to abnormal alpha synuclein, a brain protein that is abnormal in diseases like Parkinson’s Disease and Multiple System Atrophy (MSA). The purpose of this research to see how well FD4 can bind to abnormal alpha synuclein protein in the brain of people with and without Parkinson’s disease and MSA.

Disclaimer: This research opportunity is not affiliated with NAF. We are sharing this information as a resource for the Ataxia community. Sharing this information does not imply endorsement by NAF. 

About the Study

Eligible Ataxia Types
Multiple System Atrophy (MSA)

Type of Study
Type Name

Clinical Trial Phase
Type Name

Age Range
Type Name

Study Start Date
01/01/2024

Estimated Completion Date
00/00/0000

IRB Approval #
Type Name

Location(s)

Name
Street Adress
City, ST 

Contact Information

Name
Email Address
Phone

What does participation in the study entail?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

What are the potential benefits for participants?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

What are the potential risks for participants?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Is there financial compensation?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Is there travel reimbursement?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Who is eligible?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Additional Information or Resources

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

View Other Research Opportunities

FD4 is a new investigational radiotracer or imaging agent that binds to abnormal alpha synuclein, a brain protein that is abnormal in diseases like Parkinson’s Disease and Multiple System Atrophy (MSA). The purpose of this research to see how well FD4 can bind to abnormal alpha synuclein protein in the brain of people with and without Parkinson’s disease and MSA.

Disclaimer: This research opportunity is not affiliated with NAF. We are sharing this information as a resource for the Ataxia community. Sharing this information does not imply endorsement by NAF. 

About the Study

Eligible Ataxia Types
Multiple System Atrophy (MSA)

Type of Study
Type Name

Clinical Trial Phase
Type Name

Age Range
Type Name

Study Start Date
01/01/2024

Estimated Completion Date
00/00/0000

IRB Approval #
Type Name

Location(s)

Name
Street Adress
City, ST 

Contact Information

Name
Email Address
Phone

What does participation in the study entail?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

What are the potential benefits for participants?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

What are the potential risks for participants?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Is there financial compensation?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Is there travel reimbursement?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Who is eligible?

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

Additional Information or Resources

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Ut elit tellus, luctus nec ullamcorper mattis, pulvinar dapibus leo.

View Other Research Opportunities

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